Recognizing GI bleeding

Where the blood shows up hints at where it came from.

Upper GI bleedAbove the ligament of TreitzHematemesis: red or coffee-groundMelena (black, tarry)Peptic ulcer, varices, gastritisBUN may riseLower GI bleedColon and rectumHematochezia (red or maroon stool)Diverticulosis, polyps, cancerHemorrhoidsOften stops on its ownvs
Upper vs lower GI bleed. NSAIDs, anticoagulants, alcohol, and H. pylori infection raise the risk of upper GI bleeding.
AirwayTurn to sideif vomitingAccessTwolarge-boreIVsLabsCBC, coags,type & crossReplaceFluids, bloodas orderedMonitorVS, output,mental status
Acute GI bleed. Expect a proton pump inhibitor and urgent endoscopy. Tachycardia and orthostatic changes often appear before hypotension.

Cirrhosis and portal hypertension

Scarring of the liver causes complications throughout the body.

CirrhosisVaricesLife-threateningbleedingAscitesInfection risk (SBP)EncephalopathyAmmonia buildupCoagulopathyHigh INR, lowplatelets
Cirrhosis complications. Jaundice, spider angiomas, palmar erythema, and edema are also common.
ProblemNursing care
Variceal bleedingTreat as an emergency; expect octreotide, IV antibiotics, and endoscopic banding; protect the airway
AscitesLow-sodium diet, diuretics, daily weight and girth (same place each time), monitor potassium and kidney function
ParacentesisVoid first; position upright; monitor BP afterward; albumin may be given after large-volume removal
Spontaneous bacterial peritonitisReport fever, abdominal pain or tenderness, or new confusion
Bleeding riskSmall-gauge needles, prolonged pressure, soft toothbrush, electric razor; vitamin K or blood products as ordered

Teach complete alcohol abstinence and avoidance of NSAIDs and over-the-counter sleep aids.

Hepatic encephalopathy

A damaged liver cannot clear ammonia, which affects the brain.

  • Cues: sleep-wake reversal, subtle personality changes, confusion, asterixis (flapping tremor of outstretched hands), fetor hepaticus, progressing to coma.
  • Triggers: GI bleeding, infection, constipation, dehydration, electrolyte imbalance (especially low potassium), sedatives, and opioids.
  • Lactulose: traps ammonia in the bowel and removes it in stool; titrate to about 2–3 soft stools daily. Report watery diarrhea, which causes dehydration and low potassium.
  • Rifaximin may be added to reduce ammonia-producing gut bacteria.
  • Nutrition: current guidance avoids routine protein restriction; small frequent meals and a late-evening snack help prevent muscle breakdown.
  • Safety: fall precautions, frequent neurologic checks, and aspiration precautions.

Peptic ulcer complications

Know when abdominal pain becomes a surgical emergency.

ComplicationKey cuesAction
BleedingCoffee-ground emesis, melena, tachycardiaIV access, labs, PPI, endoscopy
PerforationSudden severe pain, rigid boardlike abdomen, rebound tendernessNPO, notify immediately, prepare for surgery
ObstructionVomiting undigested food, fullnessNPO, NG decompression as ordered

Teach clients to take H. pylori therapy exactly as prescribed and to avoid NSAIDs and smoking.

NGN-style case

Spotting a bleed in a client with cirrhosis.

functions Clinical judgment case — new confusion

A client with alcohol-related cirrhosis has become confused and drowsy since this morning. HR 118, BP 96/58. The nurse notes black, tarry stool and asterixis. What is the most likely explanation, and what should the nurse do?

  1. Recognize cues: melena, tachycardia, low BP, new confusion, asterixis.
  2. Analyze: upper GI bleeding (possibly variceal) causing hypovolemia and triggering hepatic encephalopathy (blood in the gut raises ammonia).
  3. Priorities: protect the airway (aspiration risk), establish large-bore IV access, send labs and type and crossmatch, notify the provider urgently.
  4. Anticipate fluids and blood, octreotide, IV antibiotics, urgent endoscopy, and lactulose.

Answer. Suspect upper GI bleeding precipitating hepatic encephalopathy; protect the airway, start resuscitation, and notify the provider immediately.